CRP and myocardial infarction: The authors found that even in the absence of systemic inflammation (as measured by CRP), circulating monocytes from patients with myocardial infarction (MI) had an increased overall cytokine production capacity following ex vivo LPS stimulation, with the strongest difference reported for IL‐10, which was associated with an enrichment of H3K4me3 at the promoter region [8].