Extensive research has underscored the role of the cGAS‒STING pathway across a spectrum of diseases, such as infections,5 autoimmune disorders,7 cancers,8 fibrotic conditions,9 and neurodegenerative diseases.10, 11, 12 Inhibiting this pathway has been shown to mitigate inflammatory disease progression, suggesting its potential as a therapeutic target for autoimmune and inflammatory conditions.6 This evidence concerns the gene STING1 and infection.