The search for early AKI biomarkers has primarily focused on proteins related to the progression or development of kidney injury, such as NGAL, KIM‐1, and IL‐8.[86, 87, 88] But the existing diagnostic approaches are characterized by their invasive nature and their limitation in providing tissue‐specific details to delineate the extent and condition of pathological changes. The gene discussed is LCN2; the disease is acute kidney injury.