TF and acute kidney injury: Animal‐based research has revealed that injecting non‐bound transferrin intraperitoneally can diminish the levels of free iron in the circulation of mice that have experienced ischemia‐reperfusion, halt the formation of superoxides in the kidneys, and decrease the inflammation caused by neutrophil infiltration,[95] indicating that transferrin saturation level can serve as an early predictor of AKI after cardiac surgery, and non‐coupled transferrin can be used to enhance endogenous iron‐binding capacity.