AXL, a TAM-family receptor highly expressed during early and mid-gestation, mediates ZIKV entry via receptor-mediated endocytosis, with productive infection demonstrated in syncytiotrophoblasts, cytotrophoblasts, and Hofbauer cells; redundancy with other TAM receptors, however, indicates that AXL is not strictly essential for placental permissiveness (62–64). Here, AXL is linked to infection.