About 5–10% of ALS cases are familial ALS (fALS), primarily resulting from mutations in certain genes such as SOD1, FUS, TARDBP (TDP‐43), VAPB, MATR3 (matrin‐3), and ATXN2 (ataxin‐2) (Yousefian‐Jazi et al. This evidence concerns the gene ATXN2 and amyotrophic lateral sclerosis.