About 5–10% of ALS cases are familial ALS (fALS), primarily resulting from mutations in certain genes such as SOD1, FUS, TARDBP (TDP‐43), VAPB, MATR3 (matrin‐3), and ATXN2 (ataxin‐2) (Yousefian‐Jazi et al. Here, SOD1 is linked to amyotrophic lateral sclerosis.