Contrarily, different, perilous interrelations occurred, likely specific, for fibronectin (corneal ulcer ↑ ↔ corneal neovascularization ↑↔ intraocular pressure ↑), MMP-inhibitors (corneal ulcer ↑ ↔ corneal neovascularization ↓↔ intraocular pressure ↑ and tendon healing ↓), EGF (corneal ulcer ↑ ↔ corneal neovascularization ↑↔ intraocular pressure not affected (0), and ↑ tendon healing), and FGF (corneal ulcer ↑ ↔ corneal neovascularization ↑ ↔ intraocular pressure ↑, and ↑ tendon healing). This evidence concerns the gene EGF and corneal infection.