Moreover, AHNAK2 has been shown to exert a regulatory influence on tumor progression, suggesting its potential as a key player in the pathophysiology of PAAD.[50] Previous research[51,52] has indicated that AHNAK2 participates in the autophagy process and modulates the proliferation, invasion, migration, and ferroptosis of lung adenocarcinoma cells. This evidence concerns the gene AHNAK2 and lung adenocarcinoma.