Because of its antimicrobial, LPS-neutralizing, and wound-healing properties [37,38,39] as well as the negative correlation between cathelicidin concentration and tissue remodeling in lung fibrosis [40,41] and in advanced stages of COPD [42], maintenance of CAMP physiological concentration should be considered as a therapeutic strategy for pulmonary fibrosis. Here, CAMP is linked to chronic obstructive pulmonary disease.