After adjustment for a broad panel of clinical, hormonal, metabolic, and anthropometric covariates (age, BMI, gravida, parity, number of abortions, mFGS score, FSH, LH, prolactin, TSH, total testosterone, DHEA-S, lipid profile, fasting glucose, waist and hip circumferences, and CRP), the within-group correlations remained weak and statistically non-significant (PCOS: partial ρ = 0.161, p = 0.475; controls: partial ρ = −0.072, p = 0.755). This evidence concerns the gene PRL and polycystic ovary syndrome.