AKT1 and Arrhythmogenic right ventricular dysplasia: Additional pathways included glycerolipid metabolism, fatty acid biosynthesis, ECM–receptor interaction, mucin-type O-glycan biosynthesis, endocytosis, arrhythmogenic right ventricular cardiomyopathy, ubiquitin-mediated proteolysis, Rap1 signaling, and PI3K–Akt signaling (Table S1).