The key findings include significantly elevated oxidative stress biomarkers (ox-LDL, Grx activity), a pro-atherogenic lipid profile (elevated ApoB and ApoB/ApoA1 ratio, increased HDL), and a disrupted IGF-axis (significantly lower IGF-I with slightly higher IGFBP1 but no correlation between IGF-I and IGFBP-1) in patients with T1DM, despite evidence of compensatory antioxidant upregulation. This evidence concerns the gene APOA1 and type 1 diabetes mellitus.