To strengthen these observations, it will be necessary to confirm the effects of this extract in various mouse models: first, in an infectious model such as one involving H. pylori; second, in an obesity model to further assess the GLP-1 modulation observed in this study; and third, in a model of mood disorders to evaluate its potential influence on gut–brain axis communication. The gene discussed is GLP1R; the disease is obesity due to melanocortin 4 receptor deficiency.