No human cases of BOMV infection have been reported thus far, and experimental infection in type I interferon receptor knockout (IFNAR−/−) mice and HLA-A2-transgenic NOD-scid-IL-2γ receptor-knockout (NSG-A2) mice reconstituted with human hematopoiesis suggest a low pathogenic potential of BOMV in humans (27, 28) (Fig. 1A). Here, IFNAR1 is linked to infection.