CSF1R and Hodgkins lymphoma: In Hodgkin’s lymphoma, an aberrant activation of the “mammalian apparent LTR retrotransposon”(MaLR) family transposon-like human element 1B (THE1B) due to the disruption of epigenetic control by deletion of co-blocker protein CBFA2T3, promotes transcription of the non-B, myeloid-specific proto-oncogene colony-stimulating factor 1 receptor (CSF1R).