ITGAM and neoplasm: Although no single immune cell trait was universally shared across all five tumor subtypes, 12 traits were causally linked to at least two subtypes, mainly involving T cell subsets (e.g., resting Treg AC, effector memory CD4+ T cells, CD4− CD8− double‐negative T cells, CD45RA expression on TD CD8br T cells, and CD8 expression on NK/T cells), B‐cell parameters (such as IgD+ AC, CD20 expression on IgD+ CD38− naive B cells, and CD25 expression on CD4+ T cells), and myeloid/monocyte populations (including CD33dim HLA‐DR+ CD11b+ antigen–presenting cells and CD14+ CD16− monocytes).