Indeed, multiple factors such as genetic variations, processing enzymes, and RNA‐binding proteins, which are proposed to influence the post‐transcriptional maturation events of TERC, have been extensively characterized in telomere‐related diseases (TADs), including pulmonary fibrosis, dyskeratosis congenita, and aplastic anemia, in recent years (Mitchell et al. 1999; O'Connor et al. 2006; Xin et al. 2007; Chen et al. 2018; Tang et al. 2018; Xu et al. 2020; Markiewicz‐Potoczny et al. 2021). The gene discussed is TERC; the disease is dyskeratosis congenita.