Immunohistochemical staining showed that LIRI increased TGF‐β expression in AAV‐shCtl mice, while AAV‐shFabp4 treatment significantly reduced TGF‐β levels (Figure 6D,G), indicating that FABP4 knockdown attenuates pulmonary fibrosis. The gene discussed is TGFB1; the disease is pulmonary fibrosis.