These mechanisms involve its participation in regulatory networks of noncoding RNAs, its influence on cardiomyocyte apoptosis mediated by signaling pathways such as LCN2 and SIRT1, and its modulation of myocardial fibrosis pathways including TGF‐β1/Smad, RhoC, and CTGF. This evidence concerns the gene CCN2 and Myocardial fibrosis.