The positive correlation between STAT3 and CD47 expression in lung cancer was consistent with previous findings, as Kang et al. (2023) demonstrated that the α5-nAChR/STAT3/CD47 axis promotes lung adenocarcinoma progression and immune evasion, supporting a role for STAT3 as a direct regulator of CD47 expression. This evidence concerns the gene STAT3 and lung adenocarcinoma.