CVDs, such as coronary heart disease or cerebrovascular accident, frequently exhibit a number of aetiologically associated cardiometabolic risk factors, including dyslipidemia, hypertension, and elevated fasting plasma glucose, which may or may not coexist with multiple inflammatory markers (e.g., C-reactive protein (CRP), uric acid, and cytokines) and a prothrombotic state (e.g., plasminogen activator inhibitor-1) [5]. This evidence concerns the gene CRP and Stroke.