From the beginning of 3 days post-infection, autophagy was observed in the duodenal, jejunal, and ileal tissues of T. spiralis-infected mice, as demonstrated by an increase of autophagosomes and autolysosomes, elevated expression of autophagy marker proteins Beclin-1 and LC3B-II/LC3B-I, and a reduction of autophagy substrate p62 levels [8]. The gene discussed is BECN1; the disease is infection.