This could explain why FPR1, in other cancer contexts, exerts different functions: in the context of human glioblastoma, FPR1 confers a more invasive and angiogenic phenotype and modulates the tumor microenvironment to favor immunosuppression (Huang et al., 2010; Zheng et al., 2023). The gene discussed is FPR1; the disease is glioblastoma.