Furthermore, FPR1 deletion in CRC cells strongly impaired tumor-specific immune response, by reducing the ability of Dendritic cells (DCs) to migrate toward dying CRC cells (Le Naour et al., 2023), thus favoring colonic reactive hyperplasia and inflammation-induced colon tumorigenesis (Vacchelli et al., 2015; Vacchelli et al., 2016; Sztupinszki et al., 2021; Le Naour et al., 2023). The gene discussed is FPR1; the disease is neoplasm.