Prompted by the association of high PRMT1 and CAPS expression with poor survival in GBM patients (Fig. S2b,c), and based on the molecular evidence of a FOSL1-PRMT1 interaction and PRMT1-mediated regulation of CAPS, we sought to determine whether these components function as a coordinated axis to promote radioresistance and invasiveness. This evidence concerns the gene PRMT1 and glioblastoma.