Other genes, including LMO231, COL1A132, MACC133, IGF2BP134, TMPRSS235CEBPA36, JUN37and MAFB38, are involved in invasiveness, EMT, stemness, and immune microenvironment remodeling, illustrating the complex networks underlying ovarian cancer progression.Mutational analyses revealed distinct histology-specific patterns: SC showed high mutation rates in TP5339, PIK3CA40, and ZFHX341, whereas EA had higher prevalence of PTEN and ARID1A mutations42,43. The gene discussed is ARID1A; the disease is ovarian cancer.