MET and listeriosis: InlB plays a critical role in bacterial pathogenesis by exploiting host cell signaling machinery to facilitate bacterial entry.[12, 17, 18]Listeria monocytogenes, a Gram‐positive pathogen responsible for listeriosis, expresses InlB on its surface to mediate invasion into nonphagocytic cells.[19] Through high‐affinity interaction with MET, InlB hijacks host signaling pathways that ultimately support bacterial internalization and systemic infection.[12, 20, 21, 22, 23] Therefore, a complete understanding of the dynamics and mechanisms of MET activation via InlB is important.