Intrahippocampal administration of recombinant WNT3a, a canonical WNT ligand (200 ng/μl in 0.3 μL) in the contralateral dorsal DG of nerve‐injured mice, on day 10 after SNI, and then daily for 4 days, significantly enhanced sensory processing and cognitive function, as demonstrated by increased tactile thresholds (Figure 9A), improved dynamic brush stroke response scores in the ipsilateral hind paw (Figure 9B), and elevated novel object recognition indices (Figure 9C) at days 11–16 after SNI. Here, WNT3A is linked to stroke disorder.