Additionally, the enhanced levels of glucose consumption (Figure 7A), lactate production (Figure 7B), ECAR (Figure 7C), GLUT1, PDK1, LDHA, p‐PI3K, p‐AKT (Figure 7E), and reduced level of OCR (Figure 7D) in Exos‐treated NSCLC cells were partially reversed by depletion of miR‐3126‐5p. The gene discussed is AKT1; the disease is non-small cell lung carcinoma.