Moreover, γδ T cells can regulate collagen type I synthesis in lung fibroblasts both through direct cell‐to‐cell contact and through the secretion of soluble antifibrotic factors, such as IFN‐γ, highlighting a potential mechanism underlying the antifibrotic effects of γδ T cells in individuals with IPF [175]. Here, IFNG is linked to idiopathic pulmonary fibrosis.