In contrast, PLA2G7‐high macrophages facilitate FMT through the LPC/ATX/LPA/LPA2 signaling axis [121], whereas a subset of IPF‐associated macrophages (IPF‐MΦ) suppress myofibroblast regeneration via the ANXA1–FPR2 pathway [158, 159]. The gene discussed is FPR2; the disease is idiopathic pulmonary fibrosis.