With respect to alterations in the functional phenotype, IPF monocytes display prorepair M2‐like characteristics (Figure 4), including increased expression of IL‐10, CD163, and IL1R2 and decreased expression of TNF‐α and CXCL10, suggesting that monocytes in IPF patients may be more involved in aberrant tissue repair processes [109]. This evidence concerns the gene IL1R2 and idiopathic pulmonary fibrosis.