In fact, within this network, we identified interactions of NF-κB with RAB3A, CLDN6, and CLDN9. NF-κB plays a critical role in mediating responses to a notable diversity of external stimuli. It is a significant orchestrator of immune response, including specific changes of NF-κB levels resulting from parasite infections [70]. This evidence concerns the gene CLDN9 and parasitic infectious disease.