In PMP22 null mice (PMP22−/−), E‐Cadherin becomes abnormally dispersed along internodes rather than enriched at Schmidt‐Lanterman incisures (SLIs) (Neuberg et al. 1999) and approximately 15% of SLIs in 3‐month‐old HNPP model mice (PMP22+/−) were qualitatively reported to exhibit abnormal E‐Cadherin immunostaining (Hu et al. 2016). Here, CDH1 is linked to hereditary neuropathy with liability to pressure palsies.