LGI1 and encephalitis: In the acute phase, voxel‐wise 18F‐FDG PET analysis showed that patients with anti‐LGI1 encephalitis had significantly increased glucose metabolism in the hippocampus (HIP), BG, and HYP, whereas marked hypometabolism was evident in the DLPFC, precuneus (PCUN), and cingulate cortex (CC) when compared with healthy controls (Figure 2A; Figure S1).