While the transcriptomic analysis of FPW1 persister cells (Supplementary Fig. 1g) revealed several distinct GO pathways being affected by long-term CMPD1 treatment (Supplementary Fig. 1h), we found a high correlation in DEGs between RKI1 and FPW1 persisters (Fig. 1g), with 4794 overlapping DEGs (Supplementary Fig. 1i), suggesting similar transcriptomic profile in two genetically-distinct glioblastoma persister models. The gene discussed is SOX9; the disease is glioblastoma.