In contrast, the Amyg showed upregulation of genes linked to oxidative phosphorylation (Atp6v1e1, Atp5mc2, Atp6v0e2), Parkinson disease (Snca, Calm1, Slc39a10), and regulation of actin cytoskeleton (Arpc3, Nckap1, Cfl1), indicating increased mitochondrial metabolism and structural plasticity. Here, SNCA is linked to Parkinson disease.