ARPC3 and Parkinson disease: In contrast, the Amyg showed upregulation of genes linked to oxidative phosphorylation (Atp6v1e1, Atp5mc2, Atp6v0e2), Parkinson disease (Snca, Calm1, Slc39a10), and regulation of actin cytoskeleton (Arpc3, Nckap1, Cfl1), indicating increased mitochondrial metabolism and structural plasticity.