The results showed that the communication intensity of signaling pathways such as LCK, CEACAM, IFN-II, IL16, NECTIN, PTN, LAIR1, VCAM, HGF, and SPP1 increased significantly in the cervical cancer group; the communication intensity of signaling pathways such as ncWNT, CADM, RA, KIT, EGF, Histamine, NRG, CD46, and LXA4 increased significantly in the healthy control group (Figure 8b). The gene discussed is LCK; the disease is cervical cancer.