Mechanistically, tumor-derived exosomal miR-205-3p downregulates AR, promoting epithelial–mesenchymal transition (EMT) and metastatic spread (63–65), while C/EBPβ-mediated activation of kinesin family member C1 (KIFC1) drives EMT and invasiveness in AR+ TNBC (64, 66). Here, AR is linked to neoplasm.