Table 1 demonstrates that: (a) HFSCs, HSCs, and MSCs generally respond in a comparable manner to all five listed agents; and (b) IFN-γ, which induces HF-IP collapse, also drives quiescence in HFSCs and inhibits HSC and MSC proliferation, whereas agents that restore HF-IP—except for TGF-β1—tend to promote stem cell self-renewal and/or proliferation. Here, TGFB1 is linked to hydrops fetalis.