Rat derived BM‐MSC‐ex (10 μg) delivered to rat models of infarction/reperfusion led to reduced apoptosis and less pronounced abnormal cell shape exosomes induced expression of Atg13 (stimulates autophagic activity via mTOR) and suppressed Apaf1 (a modulator of mitochondrial apoptotic processes). The gene discussed is MTOR; the disease is infarction.