PTK2 and pulmonary fibrosis: Pharmacological blockade or siRNA‐mediated silencing of FAK prevents bleomycin‐induced lung fibrosis in vivo and abrogates endothelin‐1‐stimulated fibroblast adhesion, contractility and profibrotic gene expression, demonstrating that FAK serves as the central hub linking matrix adhesion to the fibrotic phenotype [338].