NFKB1 and autoimmune gastritis: H. pylori activate NF-κB signaling through CagA, thereby inducing IL-17A expression in FOXP3 T cells, leading to gastritis (152)IL-17 produces T cells capable of inducing severe autoimmune gastritis (153)IL-17 expression showed a significant increase with the severity of chronic gastritis (154)IL-17 induces IL-8 secretion by activating the ERK 1/2 MAP kinase pathway, and the released IL-8 attracts neutrophils to promote gastritis (155)