Other hereditary forms include autosomal dominant hypophosphatemic rickets (ADHR) and autosomal recessive hypophosphatemic rickets (ARHR) variants involving genes such as DMP1 and ENPP1, while acquired causes such as tumor-induced osteomalacia (TIO) are extremely rare in children but a relevant differential diagnosis in adolescents. This evidence concerns the gene DMP1 and autosomal recessive hypophosphatemic rickets.