Indeed, they evaluated 11 analytes including IL-5, ll13, periostin, eotaxin-3, thymic stromal lymphopoietin, and immunoglobulins in a pediatric cohort composed of active EoE (n = 30), EoE in remission (n = 13), and controls (n = 34) and none were elevated or depressed compared to the different groups, nor were they correlated with peak esophageal eosinophilia, endoscopic features of EoE defined quantitatively by a validated EoE endoscopic reference score (EREFS) or esophageal thickness determined by endoscopic ultrasound. This evidence concerns the gene CCL26 and eosinophilic esophagitis.