AR in the ERα-positive state exerts an anti-estrogenic, growth-inhibitory effect and antagonizes ERα signaling [37,38], while in TNBC, AR reportedly promotes the proliferation of tumor cells or metastasis [39]; thus, the role of AR in TNBC is similar to that of ERα in ERα-positive tumors, having both favorable prognostic value and a tumor-promoting effect. Here, AR is linked to neoplasm.