In our previous study, we documented that AML cells with constitutively active fusion kinase, fibroblast growth factor receptor (FGFR) Oncogene Partner 2–fibroblast growth factor receptor 1 (FOP2-FGFR1), do not express VDR, while after disruption of the fusion gene, they do [18]. This evidence concerns the gene VDR and acute myeloid leukemia.