The latest in vitro experiments by Dong et al. showed that CEP inhibited the proliferation and migration of prostate cancer cells (PC-3 and DU145) in a concentration-dependent manner and induced apoptosis, and knockout or drug inhibition of DUSP1 could partially reverse the anti-cancer effect of CEP, confirming that DUSP1 is its key mediator. This evidence concerns the gene DUSP1 and prostate cancer.