CXCR3 and synovial sarcoma: In SS, overexpression of CXCL10 promotes the infiltration of CXCR3+ Th1 cells into target organs (e.g., salivary glands), driving interferon-gamma (IFN-γ)-mediated autoimmune responses, while CXCL10 inhibition significantly alleviates glandular inflammation.