Instead, when we examined whether the secretion of IL‐10 was associated with any specific Tc population (identified by the expression of their canonical cytokines [17]), we found that a significant proportion of TNF‐, granzyme B‐ and IL‐2‐, IL‐13‐, IL‐17A‐, and IL‐21‐secreting cells were co‐producing IL‐10 in people with diabetes as compared with ND participants (Figure 5H), suggesting the acquisition of IL‐10 production capacity by most Tc types (Figure S11). The gene discussed is IL17A; the disease is diabetes mellitus.