Depending on the specific breakpoints, different BCR::ABL1 fusion proteins such as p210, p190, p230, and atypical forms can be generated, with the BCR::ABL1 p190 protein more commonly found in MPAL patients than the p210 variant.[12] There is extraordinarily little literature related to the cytogenetic and molecular abnormalities of MPAL with atypical BCR::ABL1. Here, GOLGA4 is linked to mixed phenotype acute leukemia.