[13–15] Rare BCR::ABL1 transcripts, accounting for <1% of CML cases, e19a2 (39.8%), e13a3/e14a3 (20.5%), and e1a2 (16.9%) are the 3 most frequent transcript types.[16] Liu et al reported a 62‐year‐old male with Ph+ (atypical e13a2 BCR‐ABL1 fusion protein) MPAL, which presented with recurrent and intense bone pain due to bone marrow necrosis.[15] The atypical BCR::ABL1 e13a3 fusion in our case has not been reported yet. This evidence concerns the gene ABL1 and mixed phenotype acute leukemia.