TGFBR2 and Ehlers-Danlos syndrome, vascular type: Vascular Ehlers-Danlos syndrome, resulting from mutations in the COL3A1 gene, and Loeys-Dietz syndrome, linked to TGFBR1 and TGFBR2 mutations, are known to predispose individuals to arterial dissections due to connective tissue fragility [12].