Our observation of strong tumor-specific co-expression among SNHG17, SNHG1 and PABPC1L; together with the reciprocal association between SNHG17 and RUSC1-AS1 in multivariable models suggest a model in which lncRNA networks and translation regulators act in concert to drive tumor cell phenotypes. The gene discussed is SNHG1; the disease is neoplasm.