This has been observed in the DH model, where hippocampal LTP is normal at 1 week after stroke, then progressively worsens up to 12 weeks after stroke, and is associated with a cognitive deficit.22 However, there was no relationship between the presence of hippocampal neuronal injury or changes in thalamic NeuN density and NOR performance in this study. The gene discussed is RBFOX3; the disease is stroke disorder.